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function, is also predominantly the result of parietal lobe damage, and the relationship of the apraxias to language and to each other, exposes one of the most complicated issues in behavioral neurology The theoretical aspects of agnosia, particularly those related to the disturbances of visual processing, are discussed later in the chapter Cortical Sensory Syndromes The effects of a parietal lobe lesion on somatic sensation were rst described by Verger and then more completely by Dejerine, in his monograph L agnosie corti cale, and by Head and Holmes The latter, in their important paper of 1911, noted the close interrelationships between the thalamus and the sensory cortex As pointed out on page 133, the parietal postcentral cortical defect is essentially one of sensory discrimination, ie, an impairment or loss of the sense of position and passive movement and the ability to localize tactile, thermal, and noxious stimuli applied to the body surface; to distinguish objects by their size, shape, and texture (astereognosis); to recognize gures written on the skin; to distinguish between single and double contacts (two-point discrimination); and to detect the direction of movement of a tactile stimulus In contrast, the perception of pain, touch, pressure, vibratory stimuli, and thermal stimuli is relatively intact This type of sensory defect is sometimes referred to as cortical, although it can be produced just as well by lesions of the subcortical connections Clinicoanatomic studies indicate that parietocortical lesions that spare the postcentral gyrus produce only transient somatosensory changes or none at all (Corkin et al; Carmon and Benton) The question of bilateral sensory de cits as a result of lesions in only one postcentral convolution was raised by the studies of Semmes and of Corkin and their associates In tests of pressure sensitivity, two-point discrimination, point localization, position sense, and tactile object recognition, they found bilateral disturbances in nearly half of their patients with unilateral lesions, but the de cits were always more severe contralaterally and mainly in the hand These disturbances of discriminative sensation and the subject of tactile agnosia are discussed more fully in Chap 9 Dejerine and Mouzon described another parietal sensory syn drome in which touch, pressure, pain, thermal, vibratory, and position sense are all lost on one side of the body or in a limb This syndrome, more typically the result of a thalamic lesion, may also occur with large, acute lesions (infarcts, hemorrhages) in the central and subcortical white matter of the contralateral parietal lobe; in the latter case these symptoms recede in time, leaving more subtle defects in sensory discrimination Smaller lesions, particularly ones that result from a glancing blow to the skull or a small infarct or hemorrhage, may cause a defect in cutaneous-kinesthetic perception in a discrete part of a limb, eg, the ulnar or radial half of the hand and forearm; these cerebral lesions may mimic a peripheral nerve or root lesion (Dodge and Meirowsky) Also, a pseudothalamic pain syndrome on the side deprived of sensation by a parietal lesion has been described (Biemond) In a series of 12 such patients described by Michel and colleagues, burning or constrictive pain, identical to the thalamic pain syndrome (page 141), resulted from vascular lesions restricted to the cortex The discomfort involved the entire half of the body or matched the region of cortical hypesthesia; in a few cases the symptoms were paroxysmal Head and Holmes drew attention to a number of interesting points about patients with parietal sensory defects the easy fatigability of their sensory perceptions; the inconsistency of responses to painful and tactile stimuli; the dif culty in distinguishing more. devexpress asp.net mvc pdf viewer NET, ASP.NET MVC - GitHub
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You have to set the Content-Disposition header on the response to inline public FileResult GetHTMLPageAsPDF(long empID) { string ... Within the brain, no other territory surpasses the parietal lobes in the rich variety of clinical phenomena exposed under conditions of disease Our current understanding of the effects of parietal lobe disease contrasts sharply with that of the late nineteenth century, when these lobes, in the classic textbooks of Oppenheim and Gowers, were considered to be silent areas However, the clinical manifestations of parietal lobe disease may be subtle, requiring special techniques for their elicitation; even more dif cult is the interpretation of these abnormalities of function in terms of a coherent and plausible physiology and psychology Close to the core of the complex behavioral features that arise from lesions of the parietal lobes is the problem of agnosia Allusion has already been made to agnosia in the discussion of lesions of the temporal lobes that affect language, and similar ndings occur with lesions of the occipital lobe as discussed further on In those contexts, the term refers to a loss of recognition of an entity that cannot be attributed to a defect in the primary sensory modality As a result of the extension of the term agnosia to a loss of more complex integrated functions as described below, a number of intriguing de cits arise These syndromes expose properties of the parietal lobe that have implications regarding a map of the body schema and of external topographic space, of the ability to calculate, to differentiate left from right, to write words, and other problems discussed below The fact that apraxia, an inability to carry out a commanded task despite the retention of motor and sensory birt qr code, birt ean 13, birt code 128, birt gs1 128, birt barcode generator, birt upc-a pdf viewer in asp.net web application Pdf Viewer in ASP . net - CodeProject
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